ArrayExpress Uploaderarrayexpress_sidmousenormalTBP-13Q-12TBP-13QnormalTBP-13Q-12TBP-13QnormalTBP-13Q-12TBP-13QSCA17TBP-71Q-16TBP-71QSCA17TBP-71Q-16TBP-71QSCA17TBP-71Q-16TBP-71QSCA17TBP-71Q-27TBP-71QSCA17TBP-71Q-27TBP-71QSCA17TBP-71Q-27TBP-71Q804806[u'Transcription profiles were obtained for 2-month old mice containing an expanded or normal CAG trinucleotide repeat in the coding...17994014E-MEXP-1313/profile/8773/arrayexpressuploader39diseasehuntington diseaseproteinspinocerebellar ataxiaDec.12, 2014Falsetranscription-profiling-of-mouse-cerebellum-cellsE-MEXP-1313_A-AFFY-45Transcription profiling of mouse cerebellum cells containing expanded or normal CAG trinucleotide repeats in the coding region for TATA-binding protein (TBP)Nov.21, 2014[u'Transcription profiles were obtained for 2-month old mice containing an expanded or normal CAG trinucleotide repeat in the coding region for TATA-binding protein (TBP). Three different genotypes were used : TBP-13Q (normal), TBP-71Q (71 repeats), and TBP-105Q (105 repeats). Two lines of TBP-71Q (lines 16 and 27) were used in these experiments. ', {u'br': None}, {u'br': None}, u' The 71Q and 105Q mice express a mutant version of the protein (TBP) and faithfully model the disease SCA17 (spinocerebellar ataxia-17, huntington disease-like 4, HDL4). ', {u'br': None}, {u'br': None}, u' The constructs containing mixed CAG/CAA trinucleotide repeats (and encoding polyglutamine tracts) of variable length were made using a previously described method (Michalik A et al., Biotechniques, 2001). Briefly, synthetic CAG/CAA oligonucleotides were subcloned into a cDNA construct of the normal mouse (13 CAG/CAA) TBP gene. Because of the mixed nature of the repeat, its length is stable in mitotic and meiotic transmission.']http://www.ebi.ac.uk/arrayexpress/experiments/E-MEXP-1313http://www.ebi.ac.uk/arrayexpress/experiments/E-MEXP-1313/samples/