ArrayExpress Uploader0mousep53-/- mouse fetal liver progenitor cellsnon-transformedp53-/- mouse fetal liver progenitor cells transformed with H-RasV12H-RasV12p53-/- mouse fetal liver progenitor cells transformed with myr-Aktmyr-Aktp53-/- mouse fetal liver progenitor cells transformed with c-Mycc-Myc7160arrayexpress_sid6The activation of different oncogenic signals may primarily contribute to the heterogeneity of cancer cells. However, the exact...E-GEOD-43991/profile/8773/arrayexpressuploader24cancercellliverliver cellDec.12, 2014Falsedifferentially-expressed-genes-between-oncogene-trE-GEOD-43991_A-AFFY-45Differentially expressed genes between oncogene-transformed and non-transformed fetal liver progenitor cellsNov.12, 2014The activation of different oncogenic signals may primarily contribute to the heterogeneity of cancer cells. However, the exact mechanisms underlying different oncogenic transformation are still unclear. We used the c-Myc, H-Ras and Akt transformed liver cell model to define mRNA expression profiles in the non-transformed and the three types of oncogene-transformed cells Purified p53-/- mouse fetal liver progenitor cells were transformed by three oncogenic genetic factors, c-Myc, H-RasV12 and myristoylated-AKT1(myr-Akt). The gene expression profilings of transformed cells and control cells were analyzed through microarray.http://www.ebi.ac.uk/arrayexpress/experiments/E-GEOD-43991http://www.ebi.ac.uk/arrayexpress/experiments/E-GEOD-43991/samples/