Dataset: Role of REST in the pathogenesis of uterine fibroids
The loss of REST in uterine fibroids promotes aberrant gene expression and enables mTOR pathway activation We used siRNA to knockdown...
The loss of REST in uterine fibroids promotes aberrant gene expression and enables mTOR pathway activation We used siRNA to knockdown REST/ NRSF in cultured primary myometrial smooth muscle cells (SMCs) and global gene expression was analyzed using microarrays.
- Species:
- human
- Samples:
- 9
- Source:
- E-GEOD-41386
- Updated:
- Dec.12, 2014
- Registered:
- Sep.19, 2014
Factors:
(via ArrayExpress)
Sample | TREATMENT | ORGANISM PART |
---|---|---|
GSM101602 | treated with scrambled siRNA for 48h | Uterus, normal myometrium |
GSM101602 | treated with scrambled siRNA for 48h | Uterus, normal myometrium |
GSM101602 | treated with scrambled siRNA for 48h | Uterus, normal myometrium |
GSM1016024 | treated with siREST for 48h | Uterus, normal myometrium |
GSM1016024 | treated with siREST for 48h | Uterus, normal myometrium |
GSM1016024 | treated with siREST for 48h | Uterus, normal myometrium |
GSM1016027 | treated with scrambled siRNA for 48h | Uterus, Leiomyoma |
GSM1016027 | treated with scrambled siRNA for 48h | Uterus, Leiomyoma |
GSM1016027 | treated with scrambled siRNA for 48h | Uterus, Leiomyoma |