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<biogps><data><item key="owner">ArrayExpress Uploader</item><item key="pop_total">0</item><item key="species">human</item><item key="factors"><item><item key="GSM1005596"><item key="TREATED WITH">none (control)</item></item></item><item><item key="GSM1005596"><item key="TREATED WITH">none (control)</item></item></item><item><item key="GSM1005596"><item key="TREATED WITH">none (control)</item></item></item><item><item key="GSM1005599"><item key="TREATED WITH">5 ng/ml human recombinant BMP9 for 24hrs</item></item></item><item><item key="GSM1005599"><item key="TREATED WITH">5 ng/ml human recombinant BMP9 for 24hrs</item></item></item><item><item key="GSM1005599"><item key="TREATED WITH">5 ng/ml human recombinant BMP9 for 24hrs</item></item></item></item><item key="id">4579</item><item key="ownerprofile_id">arrayexpress_sid</item><item key="platform">4</item><item key="summary_wrapped">BMP9 signaling has been implicated in hereditary hemorrhagic telangiectasia and vascular remodeling, acting via the HHT target genes,...</item><item key="geo_gse_id">E-GEOD-40960</item><item key="owner_profile">/profile/8773/arrayexpressuploader</item><item key="factor_count">1</item><item key="sample_count">6</item><item key="tags"><item>chemokine</item><item>hereditary hemorrhagic telangiectasia</item><item>telangiectasia</item></item><item key="lastmodified">Dec.12, 2014</item><item key="is_default">False</item><item key="geo_gds_id"/><item key="slug">expression-data-from-bmp9-treated-human-dermal-mic</item><item key="geo_id_plat">E-GEOD-40960_A-AFFY-44</item><item key="name">Expression data from BMP9-treated human dermal microvascular endothelial cells (HMVEC-D)</item><item key="created">Sep.19, 2014</item><item key="summary">BMP9 signaling has been implicated in hereditary hemorrhagic telangiectasia and vascular remodeling, acting via the HHT target genes, endoglin and ALK1. This study sought to identify endothelial BMP9-regulated proteins that could affect the HHT phenotype. Gene ontology analysis of cDNA microarray data obtained following BMP9 treatment of primary human endothelial cells indicated regulation of chemokine, adhesion, and inflammation  pathways. The sample set is comprised of three biological replicate control human dermal microvascular endothelial cells, and three treated (5 ng/ml human recombinant BMP9) biological replicate human dermal microvascular endothelial cells</item><item key="source">http://www.ebi.ac.uk/arrayexpress/experiments/E-GEOD-40960</item><item key="sample_source">http://www.ebi.ac.uk/arrayexpress/experiments/E-GEOD-40960/samples/</item></data></biogps>
