<?xml version="1.0" encoding="ASCII"?>
<biogps><data><item key="owner">ArrayExpress Uploader</item><item key="pop_total">0</item><item key="species">mouse</item><item key="factors"><item><item key="GSM859567"><item key="GENOTYPE">WT</item></item></item><item><item key="GSM859567"><item key="GENOTYPE">WT</item></item></item><item><item key="GSM859567"><item key="GENOTYPE">WT</item></item></item><item><item key="GSM859570"><item key="GENOTYPE">Ezh2-null</item></item></item><item><item key="GSM859570"><item key="GENOTYPE">Ezh2-null</item></item></item><item><item key="GSM859570"><item key="GENOTYPE">Ezh2-null</item></item></item><item><item key="GSM859570"><item key="GENOTYPE">Ezh2-null</item></item></item><item><item key="GSM859574"><item key="GENOTYPE">Eed-null</item></item></item><item><item key="GSM859574"><item key="GENOTYPE">Eed-null</item></item></item><item><item key="GSM859574"><item key="GENOTYPE">Eed-null</item></item></item></item><item key="id">6744</item><item key="ownerprofile_id">arrayexpress_sid</item><item key="platform">6</item><item key="summary_wrapped">We evaluated gene expression changes in murine leukemia caused by retroviral overexpression of MLL-AF9. We compared wild-type (WT)...</item><item key="geo_gse_id">E-GEOD-34959</item><item key="owner_profile">/profile/8773/arrayexpressuploader</item><item key="factor_count">1</item><item key="sample_count">10</item><item key="tags"><item>leukemia</item></item><item key="lastmodified">Dec.12, 2014</item><item key="is_default">False</item><item key="geo_gds_id"/><item key="slug">expression-profiling-of-primary-wild-type-wt-ezh2</item><item key="geo_id_plat">E-GEOD-34959_A-AFFY-45</item><item key="name">Expression profiling of primary wild type (WT), Ezh2-null and Eed-null murine MLL-AF9 AML</item><item key="created">Nov.12, 2014</item><item key="summary">We evaluated gene expression changes in murine leukemia caused by retroviral overexpression of MLL-AF9. We compared wild-type (WT) leukemia cells with mutant leukemia cells after cre-mediated inactivation of homozygous conditional alleles for Ezh2 or Eed, both of which are components of the Polycomb Repressive Complex2. For WT cells, 3 biological replicates were hybridized. For Ezh2-null cells, 4 biological replicates were hybridized. For Eed-cells, 3 biological replicates were hybridized.</item><item key="source">http://www.ebi.ac.uk/arrayexpress/experiments/E-GEOD-34959</item><item key="sample_source">http://www.ebi.ac.uk/arrayexpress/experiments/E-GEOD-34959/samples/</item></data></biogps>
