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<biogps><data><item key="owner">ArrayExpress Uploader</item><item key="pop_total">0</item><item key="species">mouse</item><item key="factors"><item><item key="GSM709686"><item key="GENOTYPE">Wild type</item></item></item><item><item key="GSM709686"><item key="GENOTYPE">Wild type</item></item></item><item><item key="GSM709686"><item key="GENOTYPE">Wild type</item></item></item><item><item key="GSM709689"><item key="GENOTYPE">ApoD knock-out</item></item></item><item><item key="GSM709689"><item key="GENOTYPE">ApoD knock-out</item></item></item><item><item key="GSM709689"><item key="GENOTYPE">ApoD knock-out</item></item></item><item><item key="GSM709692"><item key="GENOTYPE">Human-ApoD transgenic gain of function mutant</item></item></item><item><item key="GSM709692"><item key="GENOTYPE">Human-ApoD transgenic gain of function mutant</item></item></item><item><item key="GSM709692"><item key="GENOTYPE">Human-ApoD transgenic gain of function mutant</item></item></item><item><item key="GSM709695"><item key="GENOTYPE">wild-type</item></item></item><item><item key="GSM709695"><item key="GENOTYPE">wild-type</item></item></item><item><item key="GSM709695"><item key="GENOTYPE">wild-type</item></item></item><item><item key="GSM709689"><item key="GENOTYPE">ApoD knock-out</item></item></item><item><item key="GSM709689"><item key="GENOTYPE">ApoD knock-out</item></item></item><item><item key="GSM709689"><item key="GENOTYPE">ApoD knock-out</item></item></item><item><item key="GSM709692"><item key="GENOTYPE">Human-ApoD transgenic gain of function mutant</item></item></item><item><item key="GSM709692"><item key="GENOTYPE">Human-ApoD transgenic gain of function mutant</item></item></item><item><item key="GSM709692"><item key="GENOTYPE">Human-ApoD transgenic gain of function mutant</item></item></item></item><item key="id">8544</item><item key="ownerprofile_id">arrayexpress_sid</item><item key="platform">8</item><item key="summary_wrapped">The lipocalin Apolipoprotein D (ApoD), known to protect the nervous system against oxidative stress (OS) in model organisms, is up-...</item><item key="geo_gse_id">E-GEOD-28643</item><item key="owner_profile">/profile/8773/arrayexpressuploader</item><item key="factor_count">1</item><item key="sample_count">18</item><item key="tags"><item>brain</item><item>cerebellum</item><item>nervous system</item></item><item key="lastmodified">Dec.12, 2014</item><item key="is_default">False</item><item key="geo_id_plat">E-GEOD-28643_A-AFFY-36</item><item key="slug">apod-modulation-mouse-cerebellar-transcriptome</item><item key="geo_gds_id"/><item key="name">ApoD modulation mouse cerebellar transcriptome</item><item key="created">Nov.24, 2014</item><item key="summary">The lipocalin Apolipoprotein D (ApoD), known to protect the nervous system against oxidative stress (OS) in model organisms, is up-regulated early in the mouse brain in response to the ROS generator paraquat (PQ). However, the processes triggered by this up-regulation have not been explored. We present here a study of the effect of ApoD on the early transcriptional changes upon OS in the mouse cerebellum using microarray profiling. ApoD-KO and transgenic mice over-expressing ApoD in neurons are compared to wild-type controls.</item><item key="source">http://www.ebi.ac.uk/arrayexpress/experiments/E-GEOD-28643</item><item key="sample_source">http://www.ebi.ac.uk/arrayexpress/experiments/E-GEOD-28643/samples/</item></data></biogps>
